I copied this from a PDF document from Dr. Joel Wallach's book "lets play doctor" published in 1998. This section was found in Chapter 10. Sorry if some of it is jumbled. I can send you the original PDF if you would like, just email me at
chooselife828@gmail.com.
CYSTIC FIBROSIS (mucoviscidosis): is the "crime" of
the century second only to diabetes (and that is only be-
cause diabetes affects millions and CF "only affects thou-
sands each year") in that it is preventable, 100% curable
in the early stages and can be far better managed in
chronic cases than it is currently managed by "orthodox"
medicine. Cystic fibrosis is an important fatal disease of
humans. CF was originally thought to be limited to white
populations of central European origin: today, CF has
been diagnosed in all peoples of the earth.
CF is thought to be genetically transmitted by the "or-
thodox" pediatricians, yet "they" have failed to prove
their theory despite multimillions of dollars spent in re-
search. Classically, the diagnosis is made when any two
of four criteria are present (Table 10-1), yet most "ortho-
dox" pediatricians will not diagnose CF without a posi-
tive "sweat test" (elevated level of sodium, chloride and
potassium in the sweat - greater than 65 mEq/L.
The "sweat test" has been elevated by dogma to "the
diagnostic test" for CF yet there are at least 17 known
diseases and syndromes that can give a positive sweat
test (Table 10-2), leading at least one group of investiga-
tors to refer to CF as a syndrome rather than a disease.
Initially described in 1933, CF was first thought to be the
result of a vitamin A deficiency in children dying with
celiac disease. In 1938, the term "cystic fibrosis" was
coined because the pathologist mistakenly thought the
changes in the pancreas were true cysts (fluid filled spaces
lined with normal tissue). It is well known today that
the "cysts" of CF are, in fact, a dilation of the pancreatic
functional unit (acini) with atrophy (shrinking) of the lin-
ing tissue. In 1952, the fact that congenital CF occurred
in a significant number of CF patients was established.
The foundation of the genetic theory of CF transmission
is based on the frequent congenital appearance and two
very poor papers, one published in 1913 which claimed
that two children with diarrhea had an "inborn error in
fat metabolism" and one in 1965 that did an epidemio-
logical study of a group of 232 Australian families with
CF - despite six sets of twins, the study failed to shed
clear light on the proposed genetic theory. These papers
were so poor they would not get past the letter opener at
any "orthodox" medical journal today. We have spent an inordinate amount of time on CF because this syn-
dromeagaindemonstratesveryclearlythatifanymedi-
cal specialty will be eliminated by discovery, that dis-
covery will never be given to the public by the "ortho-
dox" doctors!!!
In 1978, the first .universally accepted diagnosis of CF in
a laboratory ammal was made by one of us (Dr. Joel
changes in the pancreas and liver in baby monkeys and
were confirmed by CF experts from Johns Hopkins
Wallach). ThediagnosiswasbasedoncharacteristicCF
School of Medicine, Emory University and the Univer-
sity of Chicago! Experts from NIH and the CF Founda-
tion were overjoyed - that is until they learned that one
of us (Wallach) could reproduce the CF changes with a
congenital selenium deficiency in almost any animal spe-
cies. With this revelation, Wallach was fired with 24 hours
notice and "blackballed" from research (to show you how
ruthless they are, Wallach was fired ten days after his
wife died of cancer).
"
It has been learned recently that the positive "sweat test"
is the result of an essential fatty acid deficiency that
causes a secondary deficiency of "prostaglandin" (very
short lived hormones) that control the sodium, chlorides
and potassium levels of the sweat!!! Remember the talk
by the distinguished
anthropologist,
Dr. Johnathon
Leaky, Sr. who said "the more facts you have, the better
the truth you have."
The prevention of CF has been accomplished in pet, farm
and laboratory animals by the veterinary profession by
assuring adequate levels of selenium and essential fatty
acid nutriture to the preconception, pregnant and nurs-
ing mother. This is not as easy as it sounds because of
malabsorption problems (Le., celiac diseases and Crohn's
Disease) in a percentage of women!!! All things being
normal a supplementation of 200 mcg selenium per day and 5 gm of flaxseed oil t.i.d. would be adequate to pre-
vent CF.
Treatment of CF is very basic - treat the infant as early
as possible with selenium 1Mat 10-25 mcg per day. Plant
derived colloidal minerals may be used orally thereaf-
ter. Provide 5 gm of flaxseed oil orally t.i.d. Most impor-
tantly YOU MUST DETERMINE IF THE INFANT ISAL-
LERGIC TO WHEAT, COW'S MILK OR SOY!!! If you
do not correct the malabsorption problem, treatment will
only be minimally effective. In the case of older CF pa-
tients, IV essential fatty acids and 1M selenium provide
excellent management leading to a normal life expect-
ancy of 75 years!!! Compare this approach to the heart
and lung transplant offered by the "orthodox" pediatri-
cians!!! If the proper treatment is carried out, the "typi-
cal CF lung disease" will not develop. The lungs of CF
patients
are normal at birth and only develop
bronchiectasis after chronic essential fatty acid and cop-
per deficiencies have taken their toll. Don't forget the
base line nutritional supplementation here!
We went to China in 1988 to study Keshan Disease, a
known selenium deficiency disease of Chinese children.
We studied 1,700 autopsies and found 595 cases or 35%
had pancreatic CF (remember CF is supposed to be "ge-
netic disease of children of middle European extract" -
to justify this finding the proponents of the genetic theory
will no doubt claim that a very virile English missionary
impregnated 125,000 Chinese girls and, unfortunately,
he was "carrying the gene for CF."
Table 10-1. Four Criteria For The Diagnosis
of Cystic Fibrosis
1. Exocrine Pancreatic Insufficiency
2. Bronchiectasis
3. Positive Sweat Test
4. Family History of the Disease
Table 10-2. Diseases and Symptoms That Have
Reported Positive Sweat Test (1, 3, 9, 68, 72)
1. Adrenal Insufficiency
2. Ectodermal Dysplasia
3. Nephrogenic Diabetes Insipidus
4. Glucose-6 Phosphate Deficiency
5. Pupillatonia / Autonomic Dysfunction
6. Allergies
7. Calcifying Pancreatitis
8. Anorexia Nervosa
9. Cystic Fibrosis
10. Focal Hepatic Cirrhosis
11. Derangement of Prostaglandin Metabolism
12. Hypothyroidism
13, Fucosidosis
14. Malnutrition
15. Kwashiorkor
16. Diabetes
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